Dipeptide Diaminobutyroyl

Dipeptide Diaminobutyroyl is the proprietary name of a peptide that is also referred to as tripeptide-3 or dipeptide diaminobutyroyl benzylamide diacetate. What makes Syn-AKE so interesting is that it is a protein fragment from the venom of the Malaysian Temple Viper. It is able to reduce signal transmission between nerves and muscles.

Anti wrinkle and anti-aging, improve skin quality, face, neck and hand care products. Can be added to beauty and skin care products, such as lotion, facial mask, morning and evening cream, eye essence, etc

Dipeptide Diaminobutyroyl  is a synthetic peptide or syn-peptide. Syn peptides are small synthetic proteins that are modeled off a non-synthetic or real-world peptide. In the case of Syn-ake, it is a synthetic peptide that is modeled off a protein found in the venom of the Temple Viper. The peptide that Syn-ake mimics is Waglerin-1. Waglerin-1 prevents the uptake of sodium by the muscles by working on the mnAchR receptor. Preventing the uptake of sodium inhibits the transmission of nerve impulses to the muscles, and the muscles stay relaxed. This relaxation of the muscles, much like Botox, reduces the appearance of fine lines and wrinkles.

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What is this product?

SYN-AKE acts as an effective anti-wrinkle active product mainly by relaxing facial muscles. SYN-AKE acts on the postsynaptic membrane and is a reversible antagonist of muscle nicotinic acetylcholine receptors. SYN-AKE binds to subunits of nicotinic acetylcholine receptors, thereby blocking the binding of acetylcholine to the receptor, resulting in persistent receptor blockade. In the closed state, sodium ions cannot be ingested, cannot be depolarized, and nerve excitation transmission is blocked, so the muscles at the wrinkles are relaxed, thereby reducing wrinkles, and at the same time increasing the moisture and nutrition of the skin to protect the skin in an all-round way.

Application

SYN-AKE acts as an effective smoothing and wrinkle active by relaxing facial muscles in a manner consistent with the neuromuscular blocking compound Wagelrin 1 of Temple Viper.SYN-AKE acts on the postsynaptic membrane and is a reversible antagonist of the muscle nicotinic acetylcholine receptor (nmAChR). 
SYN-AKE combines the ε subunit of nmAChR to block the binding of acetylcholine to the receptor, which eventually leads to receptor blockade. In the closed state, sodium ions cannot be ingested, can not be depolarized, nerve excitation is blocked, and muscles are relaxed.